Your privacy, your choice

We use essential cookies to make sure the site can function. We also use optional cookies for advertising, personalisation of content, usage analysis, and social media.

By accepting optional cookies, you consent to the processing of your personal data - including transfers to third parties. Some third parties are outside of the European Economic Area, with varying standards of data protection.

See our privacy policy for more information on the use of your personal data.

for further information and to change your choices.
Skip to main content
Fig. 3 | Journal of Orthopaedic Surgery and Research

Fig. 3

From: Transcriptome sequencing–based analysis of the molecular mechanism underlying the effect of lncRNA AC003090.1 on osteoporosis

Fig. 3

The role of FOXP1, GSK3β and β-catenin in OP. (A) The relative expression levels of FOXP1, GSK3β and β-catenin in bone tissues of Control and OP groups obtained by high-throughput transcriptome sequencing. (B) The expression levels of AC003090.1, miR-203a-3p and FOXP1 in Cortical bone and Spongy bone tissues of OP group and Control group were detected by RT-qPCR. In OP group, n = 10; Control group, n = 10. (C) ROC curve analysis of FOXP1 level in bone tissue against OP. (D) RT-qPCR detection of MSCs and non-Mscs (Other) from Cortical bone and Spongy bone in Control group and OP group Expression levels of AC003090.1, miR-203a-3p and FOXP1 in cells. Magnetic activated cells sorted CD105+/CD166+ into MSCs. **P < 0.01; ***P < 0.001

Back to article page

Journal of Orthopaedic Surgery and Research

ISSN: 1749-799X