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Correction: Daurisoline attenuates H2O2-induced chondrocyte autophagy by activating the PI3 K/Akt/mTOR signaling pathway
Journal of Orthopaedic Surgery and Research volume 20, Article number: 471 (2025)
The Original Article was published on 27 March 2023
Correction: J Orthop Surg Res 18:248 (2023)
https://doiorg.publicaciones.saludcastillayleon.es/10.1186/s13018-023–03717-5
In this article Fig. 7 appeared incorrectly and has now been corrected in the original publication. For completeness and transparency, the old incorrect versions are displayed below.
Incorrect Fig. 7
DAS inhibits autophagy markers and apoptosis-related factors through the PI3K/AKT/mTOR signaling pathway. A–D Western blot analysis of the protein levels of p-AKT, T-AKT, p-PI3K, T-PI3K, p-mTOR and T-mTOR and the quantification of associated proteins in the blots shown. E–H western blot and quantitative correlation analysis of Beclin-1, LC3 and p62 in chondrocytes. I–L Western blot was performed to quantitatively analyze the expression of Bax, Bcl-2 and cleaved caspase-3. The values represent the mean ± SD. #p < 0.05 versus the control group. *p < 0.05, **p < 0.01, and ***p < 0.001 versus the control group
Corrected Fig. 7
DAS inhibits autophagy markers and apoptosis-related factors through the PI3K/AKT/mTOR signaling pathway. A–D Western blot analysis of the protein levels of p-AKT, T-AKT, p-PI3K, T-PI3K, p-mTOR and T-mTOR and the quantification of associated proteins in the blots shown. E–H western blot and quantitative correlation analysis of Beclin-1, LC3 and p62 in chondrocytes. I–L Western blot was performed to quantitatively analyze the expression of Bax, Bcl-2 and cleaved caspase-3. The values represent the mean ± SD. #p < 0.05 versus the control group. *p < 0.05, **p < 0.01, and ***p < 0.001 versus the control group
The original article has been corrected.
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Zhang, Y., Liu, W., Liu, Z. et al. Correction: Daurisoline attenuates H2O2-induced chondrocyte autophagy by activating the PI3 K/Akt/mTOR signaling pathway. J Orthop Surg Res 20, 471 (2025). https://doiorg.publicaciones.saludcastillayleon.es/10.1186/s13018-025-05845-6
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DOI: https://doiorg.publicaciones.saludcastillayleon.es/10.1186/s13018-025-05845-6